Lumbar Spine
Content
- General information for the reader
- The healthy lumbar spine
- Pathology of the lumbar spine
- Degenerative deformity: rotational spondylolisthesis
- Epidemiology of lumbar spine degeneration
- Narrowing of the spinal canal (lumbar spinal stenosis - LSS)
- Surgery in spinal stenosis
- Low back pain - surgery
- Diagnostic procedures
- Fusion of the lumbar spine
Each specific situation has to be evaluated on an individual basis. EuroSpine and its members cannot be held responsible for any misunderstandings (or misdiagnosis) based on the information provided here, which in no way attempts to replace a targeted evaluation.
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Specialist's opinion:
0 - 100 | |
101 - 200 | |
201 - 400 | |
More than 400 |
Orthopaedic Spine Surgeon | |
Neurosurgeon | |
Neurologist/Pain Specialist | |
Rheumatologist/Physical Medicine | |
Other |
0 - 50 | |
51 - 100 | |
101 - 200 | |
201 - 300 | |
More than 300 |
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Degenerative changes of the lumbar spine
General remarks
The main functions of the lumbar spine are to protect the spinal nerves and to facilitate most of the trunk's motion. The five lumbar vertebral bodies maintain distance and transmit loads from the thorax to the pelvis and serve as muscle attachments.
Motion and load create adaptive tissue changes during life. These changes include loss of tissue elasticity, growth of osteophytes and calcification of ligaments. As a result, the structures around the spinal canal increase in volume, thereby reducing the available space for the nerve roots in the canal or the outlets for the roots. This effect is sometimes emphasized by anterior vertebral slippage (degenerative spondylolisthesis) due to insufficiency by wear and tear of the vertebral facets.
The narrowing of the spinal canal is referred to as spinal stenosis.
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Symptoms of a narrow spinal canal
The predominant symptom of a narrow spinal canal is the so-called neurogenic claudication. This is a painful sensation occurring after a certain walking distance, or during a certain period of standing in an upright position. Typically, the pain disappears in a bending-forward position of the body, e.g. in the sitting position. Even if the most common symptom is leg pain (often in a specific area), other typical symptoms include weakness or unsteadiness in the legs after a certain walking distance. In severe cases, the patient may hardly be able to walk a few metres and is literally immobilized by this condition.Diagnostics of LSS
The most important clue to diagnosis is the patient’s history, as the physical examination may not reveal conclusive findings. Imaging confirms the suspected narrowing and defines the extent of the stenosis. Magnetic resonance and sometimes computer tomography (also combined with myelography) are the most commonly used techniques.Treatment
Treatment of symptomatic LSS can be non-operative or surgical.Non-operative: Although the narrowness in the spinal canal has its mechanical factor, some tissue swelling and oedema are often involved in (part of) the origin of symptoms. Medical treatment may positively influence these non-mechanical factors and lead to a reduction of pain.
Specialist's opinion:
Symptomatic LSS is always (>90%) a domain of non-operative treatment | |
Non-operative treatment should be carried out at least three months before surgical treatment is discussed. | |
Only if non-operative treatment is unsuccessful should surgery be indicated | |
Symptomatic LSS should always (>90%) be approached surgically | |
Other |
NSAIDs | |
Systemic steroids | |
Epidural steroids | |
Calcitonin | |
Pain medication | |
Antidepressants | |
Vitamins/roborant | |
Physical exercises |
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The decision for surgery is made at the time of diagnosis | |
Only after unsuccessful non-operative treatment | |
Timing is mainly determined by the severity of symptoms | |
Only in the presence of permanent neurological symptoms | |
Other |
Specialist's opinion:
Indirect decompression/interspinous distraction | |
Fusion without decompression | |
Laminectomy | |
Laminotomy | |
Percutaneous methods | |
Other |
Always | |
Never | |
Only for patients with instability/deformity | |
For patients with dominant back pain | |
Other |
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The list of treatment modalities is endless and includes surgery, which reflects the fact that none of these procedures are the ideal solution for the problem of low back pain.
With regard to surgery, the literature is controversial. Some papers deny the existence of an objective anatomical origin of LBP and reject surgery – regardless of the procedure chosen – as a possible treatment. Others support surgery if the diagnostic procedure leads to an identifiable anatomic source (facet/disc) of pain. Newer surgical techniques promise to improve the results by preserving segmental motion of the lumbar spine.
Specialist's opinion:
No | |
Yes | |
I am not a surgeon, but sometimes refer patients for surgery | |
I am not a surgeon and do not refer patients for surgery |
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Specialist's opinion:
Clinical examination | |
Imaging (radiographs, MRI, CT, others) | |
Facet infiltration | |
Discography | |
Nerve root infiltration | |
Epidural infiltration |
Ineffective non-operative treatment for at least three months | |
Source of pain identified (with invasive diagnostic procedures) | |
Multilevel pain (>3 levels) | |
Paucilevel (<3 segments) | |
Only in the presence of vertebral slipping | |
Only in the presence of degenerative deformity | |
If the MRI shows a “black disc” corresponding to the level of subjective pain (no discography) | |
Other |
Pain medication | |
Psychotropic medication | |
Physical exercises | |
Manual therapy/chiropractic | |
“Alternative” medicine (acupuncture/acupressure, atlas therapy etc.) | |
Psychological exercises (e.g. yoga or similar techniques) | |
Psychiatric exploration |
Specialist's opinion:
Disc replacement by prosthesis | |
Fusion | |
Posterior motion-preserving instrumentation | |
IDET | |
Other |
Fusion | |
Percutaneous treatment (ramus posterior) | |
Disc prosthesis | |
Posterior motion-preserving instrumentation | |
Other |
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Fusion is still the most frequently used technique in the spine. The complexity of the anatomy and proximity to neural structures makes it more difficult to replace parts of the spine and preserve motion. These techniques do exist today, but widespread conclusive clinical data and results are not yet available. (See also chapter “Motion preserving procedures”)
Fusion of the spine can be performed in various ways. However, the principle always remains the same: to achieve solid bony union between the fused vertebrae. The fusion mass to enhance bony union is generally the autologous bone of the patient, specially prepared allogenic bone or bone substitutes. To enhance solid union, metallic implants are generally used anteriorly, posteriorly or combined.
Patient’s own bone (pelvis/fibula) | |
Allogenic bone | |
Bone substitute | |
Morphogenetic proteins | |
Other |
Yes | |
No | |
Comment below |
Stand-alone posterior instrumentation (pedicle screws/translaminar) | |
Stand-alone anterior instrumentation (interbody cages, screws, plates) | |
Combined anterior (cages) and posterior instrumentation (combined approach or PLIF/TLIF) | |
Other |
Nerve root injury/neurological deficit | |
Cerebro-spinal fluid leakage | |
Wound healing/infection | |
Recurrence/incomplete decompression of stenosis (narrowing of the spinal canal) |
EuroSpine, April 2007